Corrections:

Xin Wan¹, Ci Cheng¹, Zhe Lin¹, Runqiu Jiang², Wei Zhao¹, Xin Yan¹, Junwei Tang², Kun Yao¹, Beicheng Sun² and Yun Chen¹

¹Department of Microbiology and Immunology, Nanjing Medical University, Nanjing, Jiangsu Province, China
²Liver Transplantation Center, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu Province, China

Published: January 31, 2022

Copyright: © 2022 Wan et al. This is an open access article distributed under the terms of the Creative Commons Attribution License (CC BY 4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

This article has been corrected: Due to errors during figure assembly, the GAPDH blots in Figures 2C and 2E are accidental duplicates of the GAPDH blots in Figure 3D. The proper Figure 2, produced using the original data, is given below. The authors declare that these corrections do not change the results or conclusions of this paper.

Original article: Oncotarget. 2015; 6:8822–8838. DOI: https://doi.org/10.18632/oncotarget.3558

Figure 2: TLRs and NLRs in LM-promoted dendritic maturation. BMDCs were collected for 24 h treatment (control, LPS, LM, and LM-HK). Messenger RNA levels of NLRs (A) and TLRs (B) were detected in each group by quantitative real-time PCR. Protein extracts were prepared and relative NLR and TLR protein levels were detected by western blot assays (C). Densitometry values relative to internal controls are displayed in the histograms (D). Activation of several signaling pathways was also analyzed by western blot (E). Summary statistics are depicted in the histogram (F). All data are presented as the mean ± SEM (^*p < 0.05, ^**p < 0.01, ^***p < 0.001).