Correction: Molecular crosstalk between ferroptosis and apoptosis: Emerging role of ER stress-induced p53-independent PUMA expression

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Oncotarget. 2018; 9:24869-24869. https://doi.org/10.18632/oncotarget.25365

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Se Hoon Hong1,*, Dae-Hee Lee2,3,*, Young-Sun Lee1, Min Jee Jo2, Yoon A Jeong2, William T. Kwon1, Haroon A. Choudry1, David L. Bartlett1 and Yong J. Lee1

1Department of Surgery, School of Medicine, University of Pittsburgh, Pittsburgh, PA 15213, USA
2Brain Korea 21 Program for Biomedicine Science, Korea University College of Medicine, Korea University, Seoul 02841, Republic of Korea
3Division of Oncology/Hematology, Department of Internal Medicine, College of Medicine, Korea University Medical Center, Korea University, Seoul 08308, Republic of Korea
*These authors have contributed equally to this work

Published: May 15, 2018

This article has been corrected: The correct Figure 2C is given below:
The authors declare that this correction does not change the results and conclusions of this paper.

Original article: Oncotarget. 2017; 8:115164-115178. DOI: https://doi.org/10.18632/oncotarget.23046.

Figure 2: Erastin promotes TRAIL-induced apoptosis. C. HCT116 cells were pretreated with erastin (10 or 50 μM) for 20 h and then exposed to TRAIL (1 ng/ml) for an additional 4 h. Cell death was determined using trypan blue exclusion assay. Whole-cell extracts were then analyzed with immunoblotting assay using indicated antibodies.

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PII: 25365