Corrections:

Prathibha Kirave^1,*, Piyush Gondaliya^1,*, Bhagyashri Kulkarni^1,*, Rakesh Rawal², Rachana Garg¹, Alok Jain¹, Kiran Kalia¹

¹ Department of Biotechnology, National Institute of Pharmaceutical Education and Research, Ahmedabad, Gujarat, India
² Department of Life Science, Gujarat University, Ahmedabad, Gujarat, India
^* These authors contributed equally to this work and are first authors

Published: November 14, 2025

Copyright: © 2025 Kirave et al. This is an open access article distributed under the terms of the Creative Commons Attribution License (CC BY 4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

This article has been corrected: In the FOXO3A blot panel (Figure 2F), the last two lines representing cis Sens +nc and cis Sens +miR-155 mimic are accidental duplicates of the last two lines representing exoRes+nc and exoRes + miR-155 mimic in the FOXO3A blot panel of Figure 4C. The actin band panel in Figure 5D accompanying the β-catenin is an accidental duplicate of the actin band panel of Figure 5B. The corrected Figures 2F and 5D, obtained using the original data, are shown below. The authors declare that these corrections do not change the results or conclusions of this paper.

Oncotarget. 2020; 11:1157–1171. DOI: https://doi.org/10.18632/oncotarget.27531.

Figure 2: (A) Binding position prediction of miR-155 with FOXO3 using TargetScan, DIANA microT-CDS and rna22 web-based tools. (B) FOXO3a luciferase activity in cis^Sens cells co-transfected with either NTC or miR-155 mimics and the cloned p-mirGLO-FOXO3a dual luciferase vector. Data are expressed as the mean +/− SD. **p < 0.01, significant difference vs. NTC group (n = 3). Two independent experiments gave similar results. Following transfection of miR-155 mimics in cis^Sens cells, miR-155 expression was validated by q-PCR at both the (C) cellular and (D) exosomal level. FOXO3a expression was measured by (E) q-PCR and (F) Western Blot. (G) Densitometr analysis of FOXO3a western blot normalized to β-actin as the loading control. Data are expressed as the mean +/− SD. * p < 0.05 and **p < 0.01. (n = 3). Two independent experiments gave similar results.

Figure 5: Exosomes were isolated from cis^Sens and cisRes cells transfected with either NTC or miR-155 mimics, and were used to treat naïve cis^Sens cells. cis^Sens cells were employed as controls and for comparison. (A) Effects of cisplatin treatment was analyzed on the migration of cisSens oral cancer cells receiving exosomes from cis Res+miR-155 mimic cells by wound assay. Left panel, Representative images of wound closure taken at 0 and 24 h after the scratch was made and cisplatin treatment initiated. Right panel, quantification of wound closure as analyzed using Image J. Western Blot expression of various EMT associated markers was measured. Densitometry analysis was carried out with β-actin as loading control. The protein markers included: (B) E-cadherin, (C) N-cadherin, and (D) β-catenin, (E) Twist and (F) Vimentin expressions were quantified by q-PCR and normalized with respect to 18S as the housekeeping gene. Data are expressed as mean ± SD. * p < 0.05 and **p < 0.01, ***p < 0.001 (n = 3). Two independent experiments gave similar results.