Correction: MicroRNA 603 acts as a tumor suppressor and inhibits triple-negative breast cancer tumorigenesis by targeting elongation factor 2 kinase

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Oncotarget. 2023; 14:908-909. https://doi.org/10.18632/oncotarget.28533

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Recep Bayraktar1,2, Martin Pichler4, Pinar Kanlikilicer1, Cristina Ivan1,4, Emine Bayraktar1,2, Nermin Kahraman1, Burcu Aslan1, Serpil Oguztuzun3, Mustafa Ulasli2, Ahmet Arslan2, George Calin1,4, Gabriel Lopez-Berestein1,4 and Bulent Ozpolat1,4

1Department of Experimental Therapeutics, The University of Texas MD Anderson Cancer Center, Houston, Texas, USA
2Department of Medical Biology, School of Medicine, Gaziantep University, Gaziantep, Turkey
3Department of Biology, Kirikkale University, Kirikkale, Turkey
4Center for RNA Interference and Non-Coding RNAs, The University of Texas MD Anderson Cancer Center, Houston, Texas, USA

Published: October 31, 2023

Copyright: © 2023 Bayraktar et al. This is an open access article distributed under the terms of the Creative Commons Attribution License (CC BY 4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

This article has been corrected: In Figure 4A, the MDA-MB-436 cell images are accidental duplicates of the MDAMB-231 images. The corrected Figure 4, produced using the original data, is shown below. The authors declare that these corrections do not change the results or conclusions of this paper.

Original article: Oncotarget. 2017; 8:11641–11658. DOI: https://doi.org/10.18632/oncotarget.14264

Figure 4: Transfection of TNBC cells with miR-603 suppresses migration and invasion of the cells in vitro. (A) Morphological changes in MDA-MB-231 and MDA-MB-436 cells after 48-h transfection with 50 nM miR-603 or control miRNA. Representative phase contrast micrographs are shown. (B) MDA-MB-231 and MDA-MB-436 cell lines that were transfected with miR-603, or miR-control or that did not undergo transfection (NT) were assessed for migration with the wound healing assay. After 72-h transfection, a wound was formed by scraping, and the area of the wound was measured at 0 and 36 h. The relative percentages of wound closure per field are shown on the right as means ± SDs. (C) the invasiveness of MDA-MB-231, MDA-MB-436 and BT-20 cells was assessed by using a matrigel transwell assay. The cells were transfected with miR-603 or miR-control or not treated (NT). After 72-h transfection, the cells were transferred to transwell chambers and incubated for 24 h. The invading cells were counted, and mean ± SDs from triplicate experiments are shown on the right (***p < 0.001).

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