Correction: MiR-129 blocks estrogen induction of NOTCH signaling activity in breast cancer stem-like cells

Guodong Xiao^1,*, Xiang Li^1,*, Gang Li^1,*, Boxiang Zhang¹, Chongwen Xu², Sida Qin¹, Ning Du¹, Jichang Wang³, Shou-Ching Tang^4,5, Jing Zhang¹, Hong Ren¹, Ke Chen⁶ and Xin Sun¹

¹Department of Thoracic Surgery and Oncology, The Second Department of Thoracic Surgery, Cancer Center, The First Affiliated Hospital of Xi’an Jiaotong University, Xi’an 710061, China
²Department of Otorhinolaryngology, The First Affiliated Hospital of Xi’an Jiaotong University, Xi’an 710061, China
³Department of Vascular and Endovascular Surgery, The First Affiliated Hospital of Xi’an Jiaotong University, Xi’an 710061, China
⁴Breast Cancer Program and Interdisciplinary Translational Research Team, Georgia Regents University Cancer Center, Augusta, Georgia 30912, United States
⁵Tianjin Medical University Cancer Institute and Hospital, Tianjin 300060, China
⁶Department of Urology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430022, China
^*These authors are considered as co-first authors

Published: September 15, 2023

This article has been corrected: In Figure 3D, the MCF-7 ‘Control’ image is an accidental duplicate of the ZR75-1 ‘Control’ image. The corrected Figure 3, produced using the original data, is shown below. The authors declare that these corrections do not change the results or conclusions of this paper.

Original article: Oncotarget. 2017; 8:103261–103273. DOI: https://doi.org/10.18632/oncotarget.21143

Figure 3: Suppressive miR-129 functions through inhibition on ESR1 and NOTCH signaling in breast cancer stem cells. (D) Enforced miR-129 expression decreased the proportion of CD44+/24− cells significantly, however NUMB inhibition induced NOTCH activation abolished the suppressive functions of miR-129.