Corrections:

Xue Yang^1,*, Lei Liang^1,2,*, Chen Zong¹, Fobao Lai¹, Pengxi Zhu³, Yu Liu⁴, Jinghua Jiang¹¹, Yang Yang¹, Lu Gao¹, Fei Ye¹, Qiudong Zhao¹, Rong Li¹, Zhipeng Han¹ and Lixin Wei¹

¹ Tumor Immunology and Gene Therapy Center, Eastern Hepatobiliary Surgery Hospital, the Second Military Medical University, Shanghai, China
² Medical College of Soochow University, Suzhou, China
³ Department of Pharmacy, Chang Hai Hospital, the Second Military Medical University, Shanghai, China
⁴ College of Art and Science, University of San Francisco, San Francisco, CA, USA
^* These authors have contributed equally to this work

Published: October 20, 2020

Copyright: © 2020 Yang et al. This is an open access article distributed under the terms of the Creative Commons Attribution License (CC BY 4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

This article has been corrected: Due to errors during figure assembly, the wrong transwell image was used for the lower left panel of Figure 6A - it is an accidental duplicate of the lower right panel of Figure 4C. The corrected Figure 6, obtained using original data, is shown below. The authors declare that these corrections do not change the results or conclusions of this paper.

Original article: Oncotarget. 2016; 7:1084–1095. DOI: https://doi.org/10.18632/oncotarget.6744.

Figure 6: Autophagy interference reversed recruitment of MSCs to KCs.(A) Transwell migration assay was performed to detect MSCs migration to KCs after autophagy interference. Migrated MSCs were stained by crystal violet(200×). KCs from Y+C mice were treated with 5μM CQ; KCs from O+C mice were treated with 100nm rapamycin. (B) The quantification of migrated MSCs of A. (C) Cytokines concentration in conditioned medium of KCs after autophagy interference was detected by bioplex assay. Y+C, young+CCl4; Y+C+CQ, young+CCl4+CQ; O+C, old+CCl4; O+C+rapamycin, old+CCl4+rapamycin. ^*P < 0.05.