Research Papers:
Long non-coding RNA MALAT1 acts as a competing endogenous RNA to promote malignant melanoma growth and metastasis by sponging miR-22
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Abstract
Wenkang Luan1,*, Lubo Li2,*, Yan Shi4,*, Xuefeng Bu3, Yun Xia1, Jinlong Wang1, Henry Siaw Djangmah1, Xiaohui Liu1, Yongping You5, Bin Xu1
1Department of Plastic Surgery, Affiliated People's Hospital of Jiangsu University, Zhenjiang, Jiangsu, China
2Department of Neurosurgery, Affiliated People's Hospital of Jiangsu University, Zhenjiang, Jiangsu, China
3Department of General Surgery, Affiliated People's Hospital of Jiangsu University, Zhenjiang, Jiangsu, China
4Department of Neurosurgery, Nanjing First Hospital, Nanjing Medical University, Nanjing, China
5Department of Neurosurgery, The First Affiliated Hospital of Nanjing Medical University, Nanjing, China
*These authors have contributed equally to this work
Correspondence to:
Wenkang Luan, email: [email protected]
Bin Xu, email: [email protected]
Keywords: MALAT1, miR-22, MMP14 and Snail, ceRNA, melanoma
Received: June 03, 2016 Accepted: August 13, 2016 Published: August 24, 2016
ABSTRACT
Long non-coding RNAs (lncRNAs) are involved in tumorigenesis. Metastasis-associated lung adenocarcinoma transcript 1 (MALAT1), an lncRNAs, is associated with the growth and metastasis of many human tumors, but its biological roles in malignant melanoma remain unclear. In this study, the aberrant up-regulation of MALAT1 was detected in melanoma. We determined that MALAT1 promotes melanoma cells proliferation, invasion and migration by sponging miR-22. MiR-22 was decreased and acted as a tumor suppressor in melanoma, and MMP14 and Snail were the functional targets of miR-22. Furthermore, MALAT1 could modulate MMP14 and Snail by operating as a competing endogenous RNA (ceRNA) for miR-22. The effects of MALAT1 in malignant melanoma is verified using a xenograft model. This finding elucidates a new mechanism for MALAT1 in melanoma development and provides a potential target for melanoma therapeutic intervention.
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PII: 11564