Correction: Tudor staphylococcal nuclease drives chemoresistance of non-small cell lung carcinoma cells by regulating S100A11

Anna Zagryazhskaya _, Olga Surova, Nadeem S. Akbar, Giulia Allavena, Katarina Gyuraszova, Irina B. Zborovskaya, Elena M. Tchevkina, Boris Zhivotovsky


Oncotarget. 2016; 7:22970. https://doi.org/10.18632/oncotarget.8805

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Present: Due to a technical error during image processing, an incorrect high resolution version of Figure 7 was included with the manuscript.

Corrected: Correct Figure 7 is provided below. Authors sincerely apologize for this oversight.

Original article: Oncotarget. 2015; 6(14): 12156-73. doi: 10.18632/oncotarget.3495

Figure 7: Silencing of S100A11 leads to increased formation of DNA strand breaks. A. Immunostaining of γH2AX in A549 cells treated as indicated (Scale bar, 10 μm); B. Cleavage of PARP and γH2AX level in A549 cells treated as indicated. GAPDH was used as loading control. C. TUNEL staining of DNA strand breaks (top image) and Hoechst 33342 (bottom image) in A549 cells treated as indicated. (Scale bar, 50 μm). The data were quantified using ImageJ software; the results are shown as the mean ± SEM of three independent experiments (arbitrary units). P < 0.05. For details see “Materials and Methods” section. All data are representative of three independent experiments.

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