Correction: A keratinocyte life cycle model identifies novel host genome regulation by human papillomavirus 16 relevant to HPV positive head and neck cancer

Michael R. Evans; Claire D. James; Oonagh Loughran; Tara J. Nulton; Xu Wang; Molly L. Bristol; Brad Windle; Iain M. Morgan

doi:10.18632/oncotarget.26962

Oncotarget

Oncotarget (a primarily oncology-focused, peer-reviewed, open access journal) aims to maximize research impact through insightful peer-review; eliminate borders between specialties by linking different fields of oncology, cancer research and biomedical sciences; and foster application of basic and clinical science.

Its scope is unique. The term "oncotarget" encompasses all molecules, pathways, cellular functions, cell types, and even tissues that can be viewed as targets relevant to cancer as well as other diseases. The term was introduced in the inaugural Editorial, Introducing Oncotarget.

As of January 1, 2022, Oncotarget has shifted to a continuous publishing model. Papers will now be published continuously within yearly volumes in their final and complete form and then quickly released to Pubmed.

Subscribe to receive alerts once a paper has been published by Oncotarget.

Impact Journals, LLC is the publisher of Oncotarget: www.impactjournals.com.

Impact Journals is a member of the Wellcome Trust List of Compliant Publishers.

Impact Journals is a member of the Society for Scholarly Publishing.

On December 23, 2022, Oncotarget server experienced a DDoS attack. As a result, Oncotarget site was inaccessible for a few hours. Oncotarget team swiftly dealt with the situation and took it under control. This malicious action will be reported to the FBI.

Corrections:

Correction: A keratinocyte life cycle model identifies novel host genome regulation by human papillomavirus 16 relevant to HPV positive head and neck cancer

PDF | How to cite

Oncotarget. 2019; 10:3312-3312. https://doi.org/10.18632/oncotarget.26962

Metrics: PDF 1167 views | ?

Michael R. Evans¹, Claire D. James¹, Oonagh Loughran¹, Tara J. Nulton¹, Xu Wang¹, Molly L. Bristol¹, Brad Windle^1,2 and Iain M. Morgan^1,2

¹ Department of Oral and Craniofacial Molecular Biology, VCU Philips Institute for Oral Health Research, Virginia Commonwealth University School of Dentistry, Richmond, VA, USA
² VCU Massey Cancer Center, Richmond, VA, USA

Published: May 14, 2019

This article has been corrected: Due to errors in document preparation, The NOKs used in the original version of this manuscript were in fact N/Tert-1 (both are TERT immortalized, with NOKs being from gingival keratinocytes and N/Tert-1 from foreskin keratinocytes); the manuscript is now corrected accordingly. None of the results or the conclusions from the manuscript are in any way altered by this different genetic background; HPV16 reprograms N/Tert-1 cells similarly to HPV16 in head and neck cancer as determined by analysis of The Cancer Genome Atlas (TCGA). The HPV16 immortalized human tonsil keratinocytes remain a bridge between the results with HPV16 positive N/Tert-1 and TCGA data. The authors apologize for this error.

Original article: Oncotarget. 2017; 8:81892–81909. DOI: https://doi.org/10.18632/oncotarget.18328.

All site content, except where otherwise noted, is licensed under a Creative Commons Attribution 4.0 License.
PII: 26962

Publication Alerts

Corrections:

Correction: A keratinocyte life cycle model identifies novel host genome regulation by human papillomavirus 16 relevant to HPV positive head and neck cancer