Oncotarget

Corrections:

Correction: An oral keratinocyte life cycle model identifies novel host genome regulation by human papillomavirus 16 relevant to HPV positive head and neck cancer

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Oncotarget. 2019; 10:3312-3312. https://doi.org/10.18632/oncotarget.26962

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Michael R. Evans1, Claire D. James1, Oonagh Loughran1, Tara J. Nulton1, Xu Wang1, Molly L. Bristol1, Brad Windle1,2 and Iain M. Morgan1,2

1 Department of Oral and Craniofacial Molecular Biology, VCU Philips Institute for Oral Health Research, Virginia Commonwealth University School of Dentistry, Richmond, VA, USA
2 VCU Massey Cancer Center, Richmond, VA, USA

Published: May 14, 2019

This article has been corrected: Due to errors in document preparation, The NOKs used in the original version of this manuscript were in fact N/Tert-1 (both are TERT immortalized, with NOKs being from gingival keratinocytes and N/Tert-1 from foreskin keratinocytes); the manuscript is now corrected accordingly. None of the results or the conclusions from the manuscript are in any way altered by this different genetic background; HPV16 reprograms N/Tert-1 cells similarly to HPV16 in head and neck cancer as determined by analysis of The Cancer Genome Atlas (TCGA). The HPV16 immortalized human tonsil keratinocytes remain a bridge between the results with HPV16 positive N/Tert-1 and TCGA data. The authors apologize for this error.

Original article: Oncotarget. 2017; 8:81892–81909. DOI: https://doi.org/10.18632/oncotarget.18328.


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PII: 26962