Correction: Molecular crosstalk between ferroptosis and apoptosis: Emerging role of ER stress-induced p53-independent PUMA expression

Se Hoon Hong; Dae-Hee Lee; Young-Sun Lee; Min Jee Jo; Yoon A. Jeong; William T. Kwon; Haroon A. Choudry; David L. Bartlett; Yong J. Lee

doi:10.18632/oncotarget.25365

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Corrections:

Correction: Molecular crosstalk between ferroptosis and apoptosis: Emerging role of ER stress-induced p53-independent PUMA expression

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Oncotarget. 2018; 9:24869-24869. https://doi.org/10.18632/oncotarget.25365

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Se Hoon Hong^1,*, Dae-Hee Lee^2,3,*, Young-Sun Lee¹, Min Jee Jo², Yoon A Jeong², William T. Kwon¹, Haroon A. Choudry¹, David L. Bartlett¹ and Yong J. Lee¹

¹Department of Surgery, School of Medicine, University of Pittsburgh, Pittsburgh, PA 15213, USA
²Brain Korea 21 Program for Biomedicine Science, Korea University College of Medicine, Korea University, Seoul 02841, Republic of Korea
³Division of Oncology/Hematology, Department of Internal Medicine, College of Medicine, Korea University Medical Center, Korea University, Seoul 08308, Republic of Korea
^*These authors have contributed equally to this work

Published: May 15, 2018

This article has been corrected: The correct Figure 2C is given below:
The authors declare that this correction does not change the results and conclusions of this paper.

Original article: Oncotarget. 2017; 8:115164-115178. DOI: https://doi.org/10.18632/oncotarget.23046.

Figure 2: Erastin promotes TRAIL-induced apoptosis. C. HCT116 cells were pretreated with erastin (10 or 50 μM) for 20 h and then exposed to TRAIL (1 ng/ml) for an additional 4 h. Cell death was determined using trypan blue exclusion assay. Whole-cell extracts were then analyzed with immunoblotting assay using indicated antibodies.

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Corrections:

Correction: Molecular crosstalk between ferroptosis and apoptosis: Emerging role of ER stress-induced p53-independent PUMA expression